A natural formulation for the maintenance of intestinal and digestive health and contributes to the maintenance of blood glucose levels. NutriKane D™ is a high quality food designed to complement a health regime which has been shown to improve outcomes in most individuals tested to date.

NutriKane D™ is a natural Australian product produced from whole sugar cane (sucrose removed) varieties grown in the Burdekin region of Northern Queensland mixed with a selection of natural fibres and inherent micronutrients from high quality whole plant sources. The patented process first removes the sugar, then shreds, dries and grinds the base cane into a powder.

NutriKane D is natural, pale colour, with consistency similar to that of flour. NutriKane D is available in retail carton of 28 sachets. Each carton is a 2 week supply at recommended doses.

How does it work?

It works by assisting intestinal health.

The natural foods used to make NutriKane contain a range of nutrients and all forms of dietary fibre that can be absorbed along the entire length of the intestinal tract. This provides slow release of key nutrients (unlike highly processed foods which are absorbed too quickly). This makes the digestive tract operate more effectively and that directly assists the body’s ability to cope with other health issues.

NutriKane D™ is high in inherent food micronutrients such as bioavailable chromium (highest levels of any foods tested), both polyphenolic and flavonoid antioxidants (3 times the antioxidants of green tea) and contains high quality sources of soluble and insoluble fibre. When NutriKane D™ is consumed with meals it has been shown to lower the GI of most foods.


Initial in-house studies, analysis of the composition of NutriKane™ and an extensive review of the literature suggests the following modes of action:


  • The product is high in bio-available chromium, which is a micronutrient that has been shown to be important to insulin sensitivity in non-diabetic individuals, and has also been proposed to be important in diabetic individuals [1,2,3]
  • The product contains a number of poly-phenolic compounds which have been shown to be effective in blood glucose maintenance, as well as being important in reducing inflammation.[4,5,6].
  • It has been shown that NutriKane D contains prebiotic fibres that promote the growth of advantageous bacteria in the gut and inhibit the growth of inflammation inducing bacteria. Prebiotics are food sources for the microorganisms found in the gut that contribute to the well-being of their host by positively altering the composition of the gut microbiome.
  • Research collaborations with Macquarie University have directly shown that gut bacteria are able to utilise the antioxidants naturally present in the whole plant ingredients of NutriKane D. Antioxidants have been shown to have dramatic positive effects on the growth of advantageous gut bacteria.
  • The product is high in fibre that has properties of both soluble and insoluble fibre (including resistant starch). High fibre diets have been shown to reduce postprandial blood glucose levels in animals and humans [7].
  • The product is high in antioxidants, which has been proposed to reduce oxidative stress in people experiencing blood glucose dysfunctions. In-house measurements of antioxidants in sugarcane fibre indicate that one dose has 6-8 times the amount of antioxidants found in green tea. This combined with the fact that new reports show a prolonged and improved benefit from antioxidants found in natural fibre sources suggest an increased benefit over purified antioxidants used in other trials [8,9]
  • ‘The Autoimmune Effect’. Latent Autoimmune Diabetes in Adults (LADA) is a form of diabetes that is suggested to occur when an autoimmune response in the subject causes destruction of insulin producing cells in the pancreas. It is still under debate as to whether the mechanism is genetic or environmental; however it is likely to be a combination of both. The form of diabetes found in dogs is analogous to LADA in humans and has been shown to originate in an inflammation response in the gut. There is also a large body of evidence to suggest that a general level of autoimmunity is initiated by poor intestinal health through the Toll-like receptor system (particularly TL-R2 and TL-R4). NutriKane™ is known to have benefits to micro-flora and intestinal health and there is scientific evidence to suggest that this translates to a reduction in inflammation. This research has passed beyond “proof of principle” in vitro work and is progressing to in vivo experiments, however the results observed so far do fit this model well. Reducing inflammation levels in the entire body will have long reaching benefits to multiple diseases, including Irritable Bowel Syndrome (IBS), autoimmune diseases and even such varied ailments as arthritis. In 2013, the company was awarded an Australian Research Council grant to investigate this phenomenon. [10,11,12].


  1. Mertz W. Chromium occurrence and function in biological systems. Physiol Rev 1969;49:163-239.
  2. Mertz W. Chromium in human nutrition: a review. J Nutr 1993;123:626-33.
  3. Mertz W. Interaction of chromium with insulin: a progress report. Nutr Rev 1998;56:174-7.
  4. Mauricio Duarte-Almeida J, Novoa AV, Linares AF, Lajolo FM, Ines Genovese M. Antioxidant activity of phenolics compounds from sugar cane (Saccharum officinarum L.) juice. Plant Foods Hum Nutr. 2006 Dec;61(4):187-92. Epub 2006 Nov
  5. Huang M, Xie Y, Chen L, Chu K, Wu S, Lu J, Chen X, Wang Y, Lai X. Antidiabetic Effect of the Total Polyphenolic Acids Fraction from Salvia miltiorrhiza Bunge in Diabetic Rats. Phytother Res. 2011 Nov 17. doi: 10.1002/ptr.3654.
  7. Klosterbuer A, Roughead ZF, Slavin J. Benefits of dietary fiber in clinical nutrition. Nutr Clin Pract. 2011 Oct;26(5):625-35.
  8. Padayachee, G. Netzel, M. Netzel, L. Day, D. Zabaras, D. Mikkelsen, M.J. Gidley, Binding of polyphenols to plant cell wall analogues – Part 1: Anthocyanins, Food Chemistry, 2012 134(1):155- 161.
  9. Maritim AC, Sanders RA, Watkins JB 3rd. Diabetes, oxidative stress, and antioxidants: a review. J Biochem Mol Toxicol. 2003;17(1):24-38. Review
  10. Sommer P, Sweeney G. Functional and mechanistic integration of infection and the metabolic syndrome. Korean Diabetes J. 2010 Apr;34(2):71-6.
  11. Vijay-Kumar M, Aitken JD, Carvalho FA, Cullender TC, Mwangi S, Srinivasan S, Sitaraman SV, Knight R, Ley RE, Gewirtz AT. Metabolic syndrome and altered gut microbiota in mice lacking Toll-like receptor 5. Science. 2010 Apr 9;328(5975):228-31.
  12. DeClue AE, Nickell J, Chang CH, Honaker A. Upregulation of proinflammatory cytokine production in response to bacterial pathogen–‐associated molecular patterns in dogs with diabetes mellitus undergoing insulin therapy. J Diabetes Sci Technol. 2012 May 1;6(3):496-502.

If you are a practitioner and would like to access these unique products and services please contact our friendly customer service team or alternatively on Freephone NZ 0800 88 44 33 or AUS 1800 177 959.

If you are a patient and would like to access these products and services please click here to request your nearest practitioner